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Question #46364

Difficulty: 8/10 | 🟢 Free

The Question

A researcher experimentally severs the parasympathetic neural inputs (vagus nerve) to the sinoatrial (SA) node of a mammalian heart while leaving the sympathetic inputs intact. Which of the following best describes the immediate physiological consequence on cardiac function?

Options

  • A.

    The heart rate decreases because the SA node requires continuous neural stimulation to maintain its auto-excitability.

  • B.

    The resting heart rate increases because the tonic inhibitory effect of acetylcholine, which normally hyperpolarizes the SA node, is removed.

  • C.

    The heart rate remains unchanged at 70-75 beats per minute because the cardiac conducting system is inherently myogenic and independent of neural control.

  • D.

    The ventricles undergo fibrillation because the AV node immediately takes over as the primary pacemaker due to the loss of vagal control.

Check Your Answer

Correct Answer

Step-by-Step Solution

Explanation

How the Examiner Thinks

Regulatory_Failure

Understand how NOT, EXCEPT, and logical constraints are used to manipulate exam results.

Concepts Behind This Question

Conducting System Sequence|Ipsilateral vs Contralateral Signaling|Vagus Nerve

Why This Question Is Tricky

Common Mistakes

Conceptual Pivot: Students often confuse 'myogenic' (the ability to generate an impulse without neural input) with being entirely independent of neural regulation.

A common misconception is that the resting heart rate of 72 bpm is the SA node's unmodulated intrinsic rate.

In reality, the intrinsic rate is higher (~100 bpm), and the resting rate is a result of continuous parasympathetic 'braking' (vagal tone).

Option C acts as a 'Common Half-Truth' trap by stating a true fact (the heart is myogenic) to support a false conclusion.

Quick Facts About This Question

Question Information

SubjectBiology
ChapterBody Fluids and Circulation - Class 11
ConceptConducting System Sequence
Difficulty8
Cognitive Load5
Target Solve Time120 sec
AccessFree