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Question #45738
The Question
A patient with severe hypovolemia exhibits hyper-activation of the Renin-Angiotensin-Aldosterone System (RAAS). How does this systemic endocrine response indirectly modulate the neural control of the micturition reflex?
Options
- A.
Angiotensin II directly stimulates the sympathetic nervous system to contract the detrusor muscle, preventing micturition.
- B.
Enhanced facultative water reabsorption delays the threshold stretching of the transitional epithelium, thereby postponing parasympathetic outflow.
- C.
Aldosterone directly inhibits the stretch receptors of the urinary bladder, preventing the initiation of the CNS reflex arc.
- D.
High levels of ADH stimulate the somatic motor nerve to maintain continuous contraction of the external urethral sphincter.
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Correct Answer
Step-by-Step Solution
Explanation
How the Examiner Thinks
Regulatory_Failure
Understand how NOT, EXCEPT, and logical constraints are used to manipulate exam results.
Concepts Behind This Question
RAAS Mechanism|Angiotensin II|Systemic Blood Pressure
Why This Question Is Tricky
Common Mistakes
Conceptual Pivot: Students often fail to connect endocrine fluid regulation (RAAS/ADH) with the mechanical/neural trigger of micturition.
The trap relies on students looking for a direct hormonal inhibition of bladder nerves or muscles, missing the NCERT-aligned indirect mechanical link: hormones reduce urine volume, which delays the physical stretch required to initiate the neural reflex arc.
Quick Facts About This Question
Question Information
| Subject | Biology |
| Chapter | Excretory Products and Their Elimination - Class 11 |
| Concept | Micturition Reflex Control |
| Difficulty | 8 |
| Cognitive Load | 5 |
| Target Solve Time | 120 sec |
| Access | Free |